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Figure 2. Artificial stimuli, resulting in abnormal spatiotemporal patterns of input to the amygdala, failed to activate the MeP and are categorized with heterospecific stimuli. a, FRAs expression was significantly greater (*p < 0.01) in the MeA ipsilateral to electrical stimulation of one vomeronasal organ than on the unstimulated side, or in electrode-implanted, unstimulated controls [two-way RM ANOVA with the following factors: side (ipsilateral, contralateral) and treatment (stimulated, unstimulated) with Tukey post hoc tests; n.s., nonsignificant]. The MeP was not activated; in fact, FRAs expression was significantly less (*p < 0.01) on the stimulated side (ANOVA, as above). b, The mGluR2 agonist LCCG-I disinhibited (increased) AOB output when infused unilaterally into the AOB, initiating significantly greater infusion-side Fos expression in the MeA (*p < 0.01) than on the control side or on either side of saline-infused animals. LCCG-I infusion produced no significant activation in the MeP or in the ACN of the olfactory amygdala (ANOVA, as above for each area).
