The Journal of Neuroscience, January 15, 2003, 23(2):1a-1a
CORRECTION
In the article "Disease Progression in a
Transgenic Model of Familial Amyotrophic Lateral Sclerosis Is Dependent
on Both Neuronal and Non-Neuronal Zinc Binding Proteins," by Krishna
Puttaparthi, William L. Gitomer, Uma Krishnan, Marjatta Son, Bhagya
Rajendran, and Jeffrey L. Elliott, which appeared on pages 8790-8796
of the October 15, 2002 issue, the preceding work of Nagano et al.
[Nagano S, Satoh M, Sumi H, Fujimura H, Tohyama C, Yanagihara T,
Sakoda S (2001) Reduction of metallothioneins promotes the disease
expression of familial amyotrophic lateral sclerosis mice in a
dose-dependent manner. Eur J Neurosci 13:1363-1370] was inadvertently
overlooked. Puttaparthi et al. independently confirmed the protective
effects of non-neuronal metallothionein-I and metallothionein-II in
mutant Cu,Zn-superoxide dismutase (SOD1)-deficient mice, but have also extended analysis with regard to motor function, neuronal counts, and
metallothionein-III-deficient mice.
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