Table 3.

Statistical results comparing day 1 sham to day 2 anodal conditions from Experiment 2a

Categoryt, p
A
    ERN amplitudet(17) = 5.01, p < 0.01
    FRN amplitudet(17) = 4.88, p < 0.01
    No-stop errorst(17) = 3.12, p < 0.01
    Posterror slowingt(17) = 3.58, p < 0.01
    Posterror accuracyt(17) = 3.66, p < 0.01
    No-stop RTt(17) = 0.63, p = 0.54
B
    ERN amplitudet(17) = 2.42, p < 0.03
    FRN amplitudet(17) = 2.34, p < 0.04
    No-stop errorst(17) = 2.38, p < 0.03
    No-stop RTt(17) = 2.25, p < 0.04
C
    SSRTt(17) = 0.94, p = 0.36
    P (respond/stop)t(17) = 1.33, p = 0.20
    P (respond/no-stop)t(17) = 1.10, p = 0.29
    LRP amplitudet(17) = 0.41, p = 0.69
    LRP latencyt(17) = 0.68, p = 0.51
    N2 amplitudet(17) = 0.32, p = 0.75
    N2 latencyt(17) = 0.36, p = 0.73
    CRN amplitudet(17) = 1.29, p = 0.22
    CRN latencyt(17) = 0.46, p = 0.65
Contralateral
    P1 amplitudet(17) = 0.66, p = 0.52
    P1 latencyt(17) = 0.21, p = 0.83
    N1 amplitudet(17) = 1.44, p = 0.17
    N1 latencyt(17) = 0.57, p = 0.58
Ipsilateral
    P1 amplitudet(17) = 1.33, p = 0.20
    P1 latencyt(17) = 0.75, p = 0.46
    N1 amplitudet(17) = 1.19, p = 0.25
    N1 latencyt(17) = 0.25, p = 0.80
  • aAll results from Experiment 2 replicate those obtained from Experiment 1, including the selective enhancement in the neural and behavioral indices of performance monitoring (A), the enhancement of neural and behavioral indices of learning (B, statistics based on learning rate parameter estimates), and the findings that numerous other neural and behavioral measures indexing other cognitive mechanisms were not significantly modulated by medial–frontal tDCS (C).