Regulation of neurotensin-containing neurons in the rat striatum. Effects of unilateral striatal lesions with quinolinic acid and ibotenic acid on neurotensin content and its binding site density
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Neurotensin receptors as modulators of glutamatergic transmission
2008, Brain Research ReviewsCitation Excerpt :The possible location of NTS1 on axon terminals suggests that NT may modulate dopaminergic neuronal transmission by acting presynaptically on receptors located on either dopaminergic or non-dopaminergic afferent terminals. The presence of NTS1 on presynaptic DA terminals in the caudate was confirmed by the findings obtained using 6-hydroxydopamine (6-OHDA) lesioned rats (Herve et al., 1986; Dilts and Kalivas, 1989; Schotte and Leysen, 1989; Masuo et al., 1990; Cadet et al., 1991). Moreover, experiments performed by lesioning intrinsic striatal neurons or cortico-striatal projections, suggest that NTS1 are not exclusively located on DA terminals (Goedert et al., 1984; Masuo et al., 1990).
Hippocalcin protects hippocampal neurons against excitotoxin damage by enhancing calcium extrusion
2007, NeuroscienceCitation Excerpt :In the present study, we injected QUIN directly into the hippocampus and restrained the dose in order to cause only partial damage in the CA1 subfield. QUIN has excitotoxic effects that are milder than those of kainic acid and ibotenic acid, NMDA and metabotropic glutamate receptor agonist, and lesions caused by QUIN spare the axons of the neurons (Schwarcz and Shoulson, 1987; Masuo et al., 1990). In our preliminary experiments, Nissl-stained pyknotic cells had already appeared in the CA1 pyramidal cell layer in +/+ mice 12 h after QUIN injection.
Antiparkinson-like effects of a novel neurotensin analog in unilaterally 6-hydroxydopamine lesioned rats
2001, European Journal of PharmacologyKynurenines in the CNS: From endogenous obscurity to therapeutic importance
2001, Progress in NeurobiologyEndogenous neurotoxins from tryptophan
2001, Toxicon