Density and distribution of excitatory amino acid receptors in the developing human fetal brain: A quantitative autoradiographic study
References (50)
- et al.
Glutamate recognition sites in human fetal brain
Neurosci. Lett.
(1988) - et al.
Development of glutamate binding sites and their regulation by calcium in rat hippocampus
Dev. Brain Res.
(1981) - et al.
Anatomical organization of excitatory amino acid receptors and their pathways
Trends Neurosci.
(1987) - et al.
Glutamate metabortropic and AMPA binding sites are reduced in Alzheimer's disease: an autoradiographic study of the hippocampus
Brain Res.
(1991) - et al.
Pharmacology of excitatory amino acid receptors mediating the stimulation of rat cerebellar cyclic GMP levels in vitro
Life Sci.
(1980) - et al.
Density and distribution of NMDA receptors in the human hippocampus in Alzheimer's disease
Brain Res.
(1986) - et al.
The ontogeny of excitatory amino acid receptors in rat forebrain. I. N-methyl-d-aspartate and quisqualate receptors
Neuroscience
(1990) - et al.
[3H]Glycine binding sites, NMDA and PCP receptors have similar distributions in the human hippocampus: An autoradiographic study
Brain Res.
(1989) - et al.
Characterization of excitatory amino acid receptor-mediated release of [3H]acetylcholine from rat striatal slices
Brain Res.
(1982) - et al.
Neurotoxicity of N-methyl-d-aspartate is markedly enhanced in developing rat central nervous system
Brain Res.
(1988)
Systemic administration of MK-801 protects against N-methyl-d-aspartate- and quisqualate-mediated neurotoxicity in perinatal rats
Neuroscience
The ontogeny of excitatory amino acid receptors in the rat forebrain. II. Kainic acid receptors
Neuroscience
Distribution of [3H]AMPA binding sites in rat brain as determined by quantitative autoradiography
Brain Res.
Development of high affinity kainate binding sites in human and rat hippocampi
Brain Res.
Transient increase of NMDA-binding sites in human hippocampus during development
Neurosci. Lett.
Neuronal migration, with special reference to developing human brain: A review
Brain Res.
The glutamate agonist quisqualic acid is neurotoxic in striatum and hippocampus of immature rat brain
Neurosci. Lett.
Hypoxia-ischemia produces focal disruption of glutamate receptors in developing brain
Dev. Brain Res.
“Epileptic” brain damage in rats induced by sustained electrical stimulation of the perforant path. I. Acute electrophysiological and light microscopic studies
Brain Res. Bull.
Transient increased density of NMDA binding sites in the developing rat hippocampus
Brain Res.
The dissociative anesthetics, ketamine and phencyclidine, selectively reduce excitation of central mammalian neurons by N-methyl-d-aspartate
Br. J. Pharmacol.
Biological differences between ischemia, hypoglycemia, and epilepsy
Ann. Neurol.
Ontogenetic development of kainate neurotoxicity correlates with glutamatergic innervation
Status marmoratus of the thalamus and striatum associated with athetosis and dystonia
Neurology
Hematogenous cells in the central nervous system of human embryos and fetuses
J. Comp. Neurol.
Cited by (49)
Epigenetics in child psychiatry
2021, Epigenetics in PsychiatryChronic subconvulsive activity during early postnatal life produces autistic behavior in the absence of neurotoxicity in the juvenile weanling period
2019, Behavioural Brain ResearchCitation Excerpt :Accordingly, early postnatal administration of monosodium glutamate in rat pups during the first week of life caused elevations in the expression of low affinity KA receptor types, GluR5 and GluR6, high affinity KA1 and KA2 types, as well as [3 H] KA binding at P21 which corresponds to the ages of our study [69]. In humans, distinct binding patterns of three main endogenous excitatory amino acid (EAA) receptor subtypes are well established early on by 20–21.5 gestational weeks suggesting over-excitation by environmental stress may begin in utero and continue ex utero, consistent with our postnatal rat pup model that reflects late in utero to neonatal periods to early childhood stages [70,71]. The excitatory/ inhibitory balance of ionic currents is also important for normal neural development [72].
Selective Neuronal Necrosis: Pathogenesis
2018, Volpe's Neurology of the NewbornHypoxic-Ischemic Injury in the Term Infant: Pathophysiology. Pathophysiology.
2018, Volpe's Neurology of the NewbornTransmitter Receptor Distribution in the Human Brain
2015, Brain Mapping: An Encyclopedic Reference