Models of the diffusional spread of nitric oxide: Implications for neural nitric oxide signalling and its pharmacological properties
References (30)
- et al.
Effect of LY83583 on relaxation induced by non-adrenergic non-cholinergic nerve stimulation and exogenous nitric oxide in the rat gastric fundus
Eur. J. Pharmac.
(1992) - et al.
Autoxidation kinetics of aqueous nitric oxide
FEBS Lett.
(1993) Glutamate, nitric oxide and cell-cell signalling in the nervous system
Trends Neurosci.
(1991)- et al.
Kinetics of nitric oxide autoxidation in aqueous solution
J. Biol. Chem.
(1994) - et al.
Direct electrochemical measurement of nitric oxide released from human platelets
Biochem. Biophys. Res. Commun.
(1993) - et al.
Biosynthesis of nitric oxide from l-arginine. A pathway for the regulation of cell function and communication
Biochem. Pharmac.
(1989) - et al.
Constitutive nitric oxide synthase from cerebellum is reversibly inhibited by nitric oxide formed from l-arginine
Biochem. Biophys. Res. Commun.
(1992) - et al.
Widespread tissue distribution, species distribution and changes in activity of Ca2+-dependent and Ca2+-independent nitric oxide synthases
FEBS Lett.
(1991) - et al.
The nitric oxide-cyclic GMP signalling pathway in rat brain
Neuropharmacology
(1993) - et al.
Characterization of Ca2+ signals induced in hippocampal CA1 neurones by the synaptic activation of NMDA receptors
J. Physiol.
(1993)
Conduction of Heat in Solids
(1986)
Synaptic activation of receptors in the Schaffer collateral-commissural pathway of rat hippo-campus
J. Physiol.
(1988)
The Mathematics of Diffusion
(1980)
Endothelium-derived relaxing factor and nitric oxide
Can. J. Physiol. Pharmac.
(1994)
Endothelium-dependent and -independent vasodilation involving cyclic GMP: relaxation induced by nitric oxide, carbon monoxide and light
Blood Vessels
(1991)
Cited by (425)
Ion channels in capillary endothelium
2020, Current Topics in MembranesCitation Excerpt :In the brain, it is unlikely that EC-secreted molecules travel far before meeting a receptor or enzyme upon which they act, which will limit their area of effect. Indeed, NO, which is unhindered by lipid membranes, has been estimated to only travel ~ 100 μm in any direction from its point source of release (Wood & Garthwaite, 1994). Therefore, given the considerable distance between capillaries and arterioles in many cases, it is unlikely that agents such as NO or PGI2 released from cECs deep within the capillary bed diffuse to trigger dilation of upstream arterioles.
Shades of gray in human white matter
2023, Journal of Comparative NeurologyStudy of disorders in regulatory spatiotemporal neurodynamics of calcium and nitric oxide
2023, Cognitive NeurodynamicsGhrelin Action in the PVH of Male Mice: Accessibility, Neuronal Targets, and CRH Neurons Activation
2023, Endocrinology (United States)
- ∗
To whom correspondence should be addressed.
Copyright © 1994 Published by Elsevier Ltd.