Pure cortical ischemia versus striatal ischemia. Circulatory, metabolic, and neuropathologic consequences
References (21)
- et al.
The response of GABAergic and cholinergic neurons to transient cerebral ischemia
Brain Res
(1982) - et al.
Specific alterations in local cerebral glucose utilization following striatal lesions
Brain Res
(1982) - et al.
Histochemical changes of brain dopamine in acute stage of cerebral ischemia in gerbils
Stroke
(1980) Cerebral metabolism in rock and roll rodents
J Cereb Blood Flow Metab
(1982)Middle cerebral artery occlusion in the young rat
Stroke
(1982)- et al.
Cerebral infarction after middle cerebral artery occlusion in progenies of spontaneously strokeprone and normal rats
Stroke
(1984) - et al.
Increased 2-deoxyglucose uptake in hippocampus, globus pallidus and substantia nigra after cerebral ischemia
Acta Neurol Scand
(1980) - et al.
Local cerebral glucose utilization during resolution of embolic experimental ischemia
- et al.
Effect of transient ischemia on monoamine levels in the cerebral cortex of gerbils
J Neurochem
(1978) - et al.
Increased local cerebral glucose utilization in the basal ganglia of the rolling mouse Nagoya
J Cereb Blood Flow Metab
(1982)
Cited by (54)
An experimental model of focal ischemia using an internal carotid artery approach
2010, Journal of Neuroscience MethodsCitation Excerpt :Animal models of cerebral ischemia represent an important contribution to both our understanding of the pathophysiology of stroke and the development of new therapies. Occlusion of the MCA is the most common model of simulating ischemic stroke in animal models (Albanese et al., 1980; Aspey et al., 1998; Hudgins and Garcia, 1970; Shigeno et al., 1985a; Tamura et al., 1981; Waltz et al., 1966). Many cerebral ischemia models in nonhuman primates (Hudgins and Garcia, 1970; Sundt and Waltz, 1966; Waltz et al., 1966), cats (O’Brien and Waltz, 1973) and rats (Albanese et al., 1980; Hirakawa et al., 1994; Longa et al., 1989; Robinson, 1981; Shigeno et al., 1985b; Tamura et al., 1981) have been developed to investigate the mechanisms of cerebral ischemic injury and the efficacy of various treatment modalities.
Reelin-deficient mice show impaired neurogenesis and increased stroke size
2006, Experimental NeurologyActive caspase-8 translocates into the nucleus of apoptotic cells to inactivate poly(ADP-ribose) polymerase-2
2002, Journal of Biological ChemistryCitation Excerpt :Studies were performed on 8-week-old male C57Bl/6 mice (25–30 g, Janvier, Le genest-St-Isles, France). Permanent focal cerebral ischemia was performed by electrocoagulation and section of the left middle cerebral artery (MCAO) according to Ref. 23 under 4% chloral hydrate anesthesia. Sham-operated animals were subjected to the same procedure without MCA electrocoagulation and section.
Topographical and quantitative assessment of white matter injury following a focal ischaemic lesion in the rat brain
1998, Brain Research Protocols