Effects of baclofen on nigral dopaminergic cell activity following acute and chronic haloperidol treatment
References (18)
- et al.
Effects of chronic haloperidol treatment on nigral dopaminergic cell activity
- et al.
The action of a GABA derivative in human spasticity
J. Neurol. Sci.
(1971) - et al.
Antidromic identification of dopaminergic and other output neurons of the rat substantia nigra
Brain Res.
(1978) - et al.
Dopamine concentrations in the rat brain following injections into the substantia nigra of baclofen, γ-aminobutyric acid, γ-hydroxybutyric acid, apomorphine and amphetamine
Neuropharmacology
(1978) - et al.
The biological activity of eland l-baclofen (Lioresal)
Eur. J. Pharmac.
(1978) - et al.
The action of baclofen on neurons of the substantia nigra and of the ventral tegmental area
Brain Res.
(1977) - et al.
The interaction of baclofen (β-(4-chlorophenyl) GABA) with GABA systems in rat brain: evidence for a releasing action
Brain Res.
(1978) - et al.
Effects of baclofen on dopamine metabolism and interaction with neuroleptic effects
Eur. J. Pharmac.
(1978) - et al.
Acute and chronic haloperidol treatment: Comparison of effects on nigral dopaminergic cell activity
Life. Sci.
(1978)
There are more references available in the full text version of this article.
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