Neuron
Volume 9, Issue 3, September 1992, Pages 441-448
Nitric oxide controls oscillatory activity in thalamocortical neurons
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2014, Progress in NeurobiologyCitation Excerpt :NO functions postsynaptically or at presynaptic nerve terminals and plays an important role in the regulation of neurotransmission (Garthwaite, 2008). HCN channels are potential downstream targets of NO/cGMP signaling in deep cerebellar nuclei neurons, trigeminal motoneurons, thalamocortical neurons and hypoglossal motoneurons (Abudara et al., 2002; Pape and Mager, 1992; Wenker et al., 2012; Wilson and Garthwaite, 2010). Using perforated-patch recording technique, it has been shown that application of NO donors induced a reversible increase in the amplitude of Ih, which was attributable to a depolarizing shift in the voltage dependence of HCN channel activation (Wenker et al., 2012; Wilson and Garthwaite, 2010).
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