MinireviewAdenosine and neuronal plasticity
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The role of dopamine D<inf>2</inf>-like receptors in a “depotentiation-like effect” of deep brain stimulation in kindled rats
2020, Brain ResearchCitation Excerpt :Second, in vivo induction of LTD is difficult in the adult rat hippocampus (Burette et al., 1997; Heynen et al., 1996; Staubli and Scafidi, 1997; Xu et al., 1997). Therefore, our data propose that a depotentiation-like effect of LFS may be the main mechanism underlying its inhibitory effects on kindled seizures; Third, two anticonvulsant agents, adenosine A1 receptors (Fredholm, 2003; Gouder et al., 2003; Pourgholami et al., 1997) and endocannabinoid CB1 receptors (Jones et al., 2010; Rizzo et al., 2009; Shafaroodi et al., 2004), are the most important neuromodulators involved in depotentiation (de Mendonca and Ribeiro, 1997; Izumi and Zorumski, 2016) and are also involved in mediating LFS’ inhibitory action on kindled seizures (Mardani et al., 2018; Mohammad-Zadeh et al., 2009). LFS’ restorative effect on learning and memory in kindled animals may also be explained by a depotentiation-like effect of LFS.