Erratum: We regret that, in the process of reproducing the art for the article “Amino Acid Signatures in the Primate Retina” (Michael Kalloniatis et al.), which appeared on pages 6807–6829 in the November 1, 1996 issue, Figures 3, 10, and 12 were printed out of focus, blurring the authors’ results. These figures are reprinted here.
Fig. 3. Taurine (Tau) immunoreactivity (sample 571) 1–3 mm from the fovea (A) and (sample M328) 8–10 mm from the fovea (B). A, Photoreceptors display the highest level of immunoreactivity, followed by bipolar cells. Müller’s cell somas are indistinguishable from bipolar cells in the middle of the inner nuclear layer (INL); however, Müller’s cell end-feet traversing the nerve fiber layer are easily identified. Note that horizontal cells (H) display low taurine immunoreactivity, whereas ganglion cells (G) are immunonegative. B, In the far periphery, Müller’s cells dominate taurine labeling patterns in the inner nuclear and ganglion cell layers. Ganglion cells and some amacrine cells are immunonegative. G, Ganglion cells.
Fig. 10. Foveal serial sections (sample M328) labeled for glutamate (A), GABA (B), taurine (C), glycine (D), aspartate (E), and glutamine (F). A G Γ1 amacrine cell (Awith arrow), an E4 ganglion cell (Gwith arrow), and an E1 bipolar cell (B with arrow) on the foveal edge are indicated. Note the disarray of the immunoreactivity in the inner nuclear layer (INL) near the foveal edge and the GABA processes in the foveal floor (identified by arrowheads in B).Hf, Henle’s fibers.
Fig. 12. Serial sections of midperipheral primate retina (sample M328) at 4–6 mm eccentricity, consecutively labeled for taurine (A), GABA (B), glutamate (C), and glycine (D). Several theme groups based on pattern recognition classification are indicated, including X1(X1) cells with their “null” signatures, G Γ1 amacrine cells (G1), Γ2(G2), and Γ4 (Γ4) amacrine cells to highlight the distinctively higher glutamate signals ofΓ2 cells (Γ2), G+E2(E2) and G−E1 (E1) bipolar cells, τ+G2 and τ−G1 amacrine cells, conventional E4(E4) ganglion cells, and a weakly γ+E6 (E6) ganglion cell. A GΓ1amacrine cell is also indicated.