Skip to main content

Main menu

  • HOME
  • CONTENT
    • Early Release
    • Featured
    • Current Issue
    • Issue Archive
    • Collections
    • Podcast
  • ALERTS
  • FOR AUTHORS
    • Information for Authors
    • Fees
    • Journal Clubs
    • eLetters
    • Submit
  • EDITORIAL BOARD
  • ABOUT
    • Overview
    • Advertise
    • For the Media
    • Rights and Permissions
    • Privacy Policy
    • Feedback
  • SUBSCRIBE

User menu

  • Log in
  • My Cart

Search

  • Advanced search
Journal of Neuroscience
  • Log in
  • My Cart
Journal of Neuroscience

Advanced Search

Submit a Manuscript
  • HOME
  • CONTENT
    • Early Release
    • Featured
    • Current Issue
    • Issue Archive
    • Collections
    • Podcast
  • ALERTS
  • FOR AUTHORS
    • Information for Authors
    • Fees
    • Journal Clubs
    • eLetters
    • Submit
  • EDITORIAL BOARD
  • ABOUT
    • Overview
    • Advertise
    • For the Media
    • Rights and Permissions
    • Privacy Policy
    • Feedback
  • SUBSCRIBE
PreviousNext
ARTICLE

Potentiation of Quantal Catecholamine Secretion by Glibenclamide: Evidence for a Novel Role of Sulphonylurea Receptors in Regulating the Ca2+ Sensitivity of Exocytosis

S. C. Taylor, E. Carpenter, M. L. Roberts and C. Peers
Journal of Neuroscience 15 July 1999, 19 (14) 5741-5749; DOI: https://doi.org/10.1523/JNEUROSCI.19-14-05741.1999
S. C. Taylor
1Institute for Cardiovascular Research, University of Leeds, Leeds LS2 9JT, United Kingdom, and
  • Find this author on Google Scholar
  • Find this author on PubMed
  • Search for this author on this site
E. Carpenter
1Institute for Cardiovascular Research, University of Leeds, Leeds LS2 9JT, United Kingdom, and
  • Find this author on Google Scholar
  • Find this author on PubMed
  • Search for this author on this site
M. L. Roberts
2Department of Physiology, University of Adelaide, Adelaide 5005, Australia
  • Find this author on Google Scholar
  • Find this author on PubMed
  • Search for this author on this site
C. Peers
1Institute for Cardiovascular Research, University of Leeds, Leeds LS2 9JT, United Kingdom, and
  • Find this author on Google Scholar
  • Find this author on PubMed
  • Search for this author on this site
  • Article
  • Figures & Data
  • Info & Metrics
  • eLetters
  • PDF
Loading

Article Information

DOI 
https://doi.org/10.1523/JNEUROSCI.19-14-05741.1999
PubMed 
10407015
Published By 
Society for Neuroscience
History 
  • Received April 12, 1999
  • Accepted April 28, 1999
  • First published July 15, 1999.
  • Version of record published July 15, 1999.
Copyright & Usage 
Copyright © 1999 Society for Neuroscience

Author Information

  1. S. C. Taylor1,
  2. E. Carpenter1,
  3. M. L. Roberts2, and
  4. C. Peers1
  1. 1Institute for Cardiovascular Research, University of Leeds, Leeds LS2 9JT, United Kingdom, and
  2. 2Department of Physiology, University of Adelaide, Adelaide 5005, Australia
View Full Text

Author contributions

Disclosures

    • Received April 12, 1999.
    • Accepted April 28, 1999.
  • This work was supported by The British Heart Foundation and The Wellcome Trust.

    Correspondence should be addressed to Dr. Chris Peers, Institute for Cardiovascular Research, University of Leeds, Leeds LS2 9JT, UK.

Online Impact

 

Article usage

Select a custom date range for the past year
E.g., 2023-01-31
to
E.g., 2023-01-31

Article usage: January 2018 to January 2023

AbstractFullPdf
Jan 2018320
Feb 2018031
Mar 2018120
Apr 2018091
May 2018180
Jun 20181132
Jul 2018040
Aug 20180240
Sep 20180300
Oct 20180310
Nov 20180421
Dec 20180242
Total 201861927
Jan 20190171
Feb 20191153
Mar 2019174
Apr 2019244
May 2019045
Jun 2019032
Jul 20190104
Aug 2019075
Oct 2019224
Nov 2019046
Dec 2019195
Total 201978243
Jan 2020121
Feb 2020041
Mar 2020012
May 2020161
Jun 2020111
Jul 2020132
Aug 2020131
Sep 20200103
Oct 2020074
Nov 2020094
Dec 20201122
Total 202065822
Jan 2021083
Feb 2021174
Mar 202131014
Apr 2021082
May 20211105
Jun 2021093
Jul 2021186
Aug 2021056
Sep 2021193
Oct 20213915
Nov 202101117
Dec 2021391
Total 20211310379
Jan 20222117
Feb 2022065
Mar 2022071
Apr 20220102
May 202201012
Jun 20220203
Jul 2022090
Aug 20220134
Sep 2022068
Oct 2022094
Nov 2022020
Dec 2022060
Total 2022210946
Jan 20231106
Total 20231106
Total35554203
Back to top

In this issue

The Journal of Neuroscience: 19 (14)
Journal of Neuroscience
Vol. 19, Issue 14
15 Jul 1999
  • Table of Contents
  • Index by author
Email

Thank you for sharing this Journal of Neuroscience article.

NOTE: We request your email address only to inform the recipient that it was you who recommended this article, and that it is not junk mail. We do not retain these email addresses.

Enter multiple addresses on separate lines or separate them with commas.
Potentiation of Quantal Catecholamine Secretion by Glibenclamide: Evidence for a Novel Role of Sulphonylurea Receptors in Regulating the Ca2+ Sensitivity of Exocytosis
(Your Name) has forwarded a page to you from Journal of Neuroscience
(Your Name) thought you would be interested in this article in Journal of Neuroscience.
CAPTCHA
This question is for testing whether or not you are a human visitor and to prevent automated spam submissions.
Print
View Full Page PDF
Citation Tools
Potentiation of Quantal Catecholamine Secretion by Glibenclamide: Evidence for a Novel Role of Sulphonylurea Receptors in Regulating the Ca2+ Sensitivity of Exocytosis
S. C. Taylor, E. Carpenter, M. L. Roberts, C. Peers
Journal of Neuroscience 15 July 1999, 19 (14) 5741-5749; DOI: 10.1523/JNEUROSCI.19-14-05741.1999

Citation Manager Formats

  • BibTeX
  • Bookends
  • EasyBib
  • EndNote (tagged)
  • EndNote 8 (xml)
  • Medlars
  • Mendeley
  • Papers
  • RefWorks Tagged
  • Ref Manager
  • RIS
  • Zotero
Respond to this article
Request Permissions
Share
Potentiation of Quantal Catecholamine Secretion by Glibenclamide: Evidence for a Novel Role of Sulphonylurea Receptors in Regulating the Ca2+ Sensitivity of Exocytosis
S. C. Taylor, E. Carpenter, M. L. Roberts, C. Peers
Journal of Neuroscience 15 July 1999, 19 (14) 5741-5749; DOI: 10.1523/JNEUROSCI.19-14-05741.1999
del.icio.us logo Digg logo Reddit logo Twitter logo CiteULike logo Facebook logo Google logo Mendeley logo
  • Tweet Widget
  • Facebook Like
  • Google Plus One

Jump to section

  • Article
    • Abstract
    • MATERIALS AND METHODS
    • RESULTS
    • DISCUSSION
    • Footnotes
    • REFERENCES
  • Figures & Data
  • Info & Metrics
  • eLetters
  • PDF

Keywords

  • glibenclamide
  • sulfonylurea
  • catecholamines
  • exocytosis
  • Ca2+
  • amperometry
  • KATPchannel
  • pheochromocytoma

Responses to this article

Respond to this article

Jump to comment:

No eLetters have been published for this article.

Related Articles

Cited By...

More in this TOC Section

  • Intracranially Administered Anti-Αβ Antibodies Reduce β-Amyloid Deposition by Mechanisms Both Independent of and Associated with Microglial Activation
  • Calcium Influx via L- and N-Type Calcium Channels Activates a Transient Large-Conductance Ca2+-Activated K+Current in Mouse Neocortical Pyramidal Neurons
  • Neural Correlates of Competing Fear Behaviors Evoked by an Innately Aversive Stimulus
Show more ARTICLE
  • Home
  • Alerts
  • Visit Society for Neuroscience on Facebook
  • Follow Society for Neuroscience on Twitter
  • Follow Society for Neuroscience on LinkedIn
  • Visit Society for Neuroscience on Youtube
  • Follow our RSS feeds

Content

  • Early Release
  • Current Issue
  • Issue Archive
  • Collections

Information

  • For Authors
  • For Advertisers
  • For the Media
  • For Subscribers

About

  • About the Journal
  • Editorial Board
  • Privacy Policy
  • Contact
(JNeurosci logo)
(SfN logo)

Copyright © 2023 by the Society for Neuroscience.
JNeurosci Online ISSN: 1529-2401

The ideas and opinions expressed in JNeurosci do not necessarily reflect those of SfN or the JNeurosci Editorial Board. Publication of an advertisement or other product mention in JNeurosci should not be construed as an endorsement of the manufacturer’s claims. SfN does not assume any responsibility for any injury and/or damage to persons or property arising from or related to any use of any material contained in JNeurosci.