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ARTICLE, Cellular/Molecular

Co-Induction of p75NTR and p75NTR-Associated Death Executor in Neurons After Zinc Exposure in Cortical Culture or Transient Ischemia in the Rat

Jeong Ae Park, Joo-Yong Lee, Taka-Aki Sato and Jae-Young Koh
Journal of Neuroscience 15 December 2000, 20 (24) 9096-9103; DOI: https://doi.org/10.1523/JNEUROSCI.20-24-09096.2000
Jeong Ae Park
1National Creative Research Initiative Center for the Study of CNS Zinc and Department of Neurology, University of Ulsan College of Medicine, 388-1 Poongnap-Dong Songpa-Gu, Seoul 138-736, Korea,
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Joo-Yong Lee
1National Creative Research Initiative Center for the Study of CNS Zinc and Department of Neurology, University of Ulsan College of Medicine, 388-1 Poongnap-Dong Songpa-Gu, Seoul 138-736, Korea,
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Taka-Aki Sato
2Molecular Oncology Laboratory, Tsukuba Life Science Center, Institute of Physical and Chemical Research (RIKEN), Ibaraki 305-0074, Japan, and
3Division of Molecular Oncology, Department of Otolaryngology, Head and Neck Surgery and Pathology, College of Physicians and Surgeons, Columbia University, New York, New York 10032
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Jae-Young Koh
1National Creative Research Initiative Center for the Study of CNS Zinc and Department of Neurology, University of Ulsan College of Medicine, 388-1 Poongnap-Dong Songpa-Gu, Seoul 138-736, Korea,
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The Journal of Neuroscience: 20 (24)
Journal of Neuroscience
Vol. 20, Issue 24
15 Dec 2000
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Co-Induction of p75NTR and p75NTR-Associated Death Executor in Neurons After Zinc Exposure in Cortical Culture or Transient Ischemia in the Rat
Jeong Ae Park, Joo-Yong Lee, Taka-Aki Sato, Jae-Young Koh
Journal of Neuroscience 15 December 2000, 20 (24) 9096-9103; DOI: 10.1523/JNEUROSCI.20-24-09096.2000

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Co-Induction of p75NTR and p75NTR-Associated Death Executor in Neurons After Zinc Exposure in Cortical Culture or Transient Ischemia in the Rat
Jeong Ae Park, Joo-Yong Lee, Taka-Aki Sato, Jae-Young Koh
Journal of Neuroscience 15 December 2000, 20 (24) 9096-9103; DOI: 10.1523/JNEUROSCI.20-24-09096.2000
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Keywords

  • neurotrophin
  • nerve growth factor
  • apoptosis
  • caspase
  • TFL-Zn
  • calcium
  • oxidative injury

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