Development/Plasticity/Repair
Apolipoprotein E Markedly Facilitates Age-Dependent Cerebral Amyloid Angiopathy and Spontaneous Hemorrhage in Amyloid Precursor Protein Transgenic Mice
John D. Fryer, Jennie W. Taylor, Ronald B. DeMattos, Kelly R. Bales, Steven M. Paul, Maia Parsadanian and David M. Holtzman
Journal of Neuroscience 27 August 2003, 23 (21) 7889-7896; DOI: https://doi.org/10.1523/JNEUROSCI.23-21-07889.2003
John D. Fryer
Departments of
1Neurology and
2Molecular Biology and Pharmacology and the
3Center for the Study of Nervous System Injury, Washington University School of Medicine, St. Louis, Missouri 63110,
4Neuroscience Discovery Research, Eli Lilly and Company, Lilly Research Laboratories, Indianapolis, Indiana 46285, and
5Departments of Pharmacology, Toxicology, and Psychiatry, Indiana University School of Medicine, Indianapolis, Indiana 46285
Jennie W. Taylor
Departments of
1Neurology and
2Molecular Biology and Pharmacology and the
3Center for the Study of Nervous System Injury, Washington University School of Medicine, St. Louis, Missouri 63110,
4Neuroscience Discovery Research, Eli Lilly and Company, Lilly Research Laboratories, Indianapolis, Indiana 46285, and
5Departments of Pharmacology, Toxicology, and Psychiatry, Indiana University School of Medicine, Indianapolis, Indiana 46285
Ronald B. DeMattos
Departments of
1Neurology and
2Molecular Biology and Pharmacology and the
3Center for the Study of Nervous System Injury, Washington University School of Medicine, St. Louis, Missouri 63110,
4Neuroscience Discovery Research, Eli Lilly and Company, Lilly Research Laboratories, Indianapolis, Indiana 46285, and
5Departments of Pharmacology, Toxicology, and Psychiatry, Indiana University School of Medicine, Indianapolis, Indiana 46285
Kelly R. Bales
Departments of
1Neurology and
2Molecular Biology and Pharmacology and the
3Center for the Study of Nervous System Injury, Washington University School of Medicine, St. Louis, Missouri 63110,
4Neuroscience Discovery Research, Eli Lilly and Company, Lilly Research Laboratories, Indianapolis, Indiana 46285, and
5Departments of Pharmacology, Toxicology, and Psychiatry, Indiana University School of Medicine, Indianapolis, Indiana 46285
Steven M. Paul
Departments of
1Neurology and
2Molecular Biology and Pharmacology and the
3Center for the Study of Nervous System Injury, Washington University School of Medicine, St. Louis, Missouri 63110,
4Neuroscience Discovery Research, Eli Lilly and Company, Lilly Research Laboratories, Indianapolis, Indiana 46285, and
5Departments of Pharmacology, Toxicology, and Psychiatry, Indiana University School of Medicine, Indianapolis, Indiana 46285
Maia Parsadanian
Departments of
1Neurology and
2Molecular Biology and Pharmacology and the
3Center for the Study of Nervous System Injury, Washington University School of Medicine, St. Louis, Missouri 63110,
4Neuroscience Discovery Research, Eli Lilly and Company, Lilly Research Laboratories, Indianapolis, Indiana 46285, and
5Departments of Pharmacology, Toxicology, and Psychiatry, Indiana University School of Medicine, Indianapolis, Indiana 46285
David M. Holtzman
Departments of
1Neurology and
2Molecular Biology and Pharmacology and the
3Center for the Study of Nervous System Injury, Washington University School of Medicine, St. Louis, Missouri 63110,
4Neuroscience Discovery Research, Eli Lilly and Company, Lilly Research Laboratories, Indianapolis, Indiana 46285, and
5Departments of Pharmacology, Toxicology, and Psychiatry, Indiana University School of Medicine, Indianapolis, Indiana 46285
In this issue
Journal of Neuroscience
Vol. 23, Issue 21
27 Aug 2003
Apolipoprotein E Markedly Facilitates Age-Dependent Cerebral Amyloid Angiopathy and Spontaneous Hemorrhage in Amyloid Precursor Protein Transgenic Mice
John D. Fryer, Jennie W. Taylor, Ronald B. DeMattos, Kelly R. Bales, Steven M. Paul, Maia Parsadanian, David M. Holtzman
Journal of Neuroscience 27 August 2003, 23 (21) 7889-7896; DOI: 10.1523/JNEUROSCI.23-21-07889.2003
Respond to this article
Apolipoprotein E Markedly Facilitates Age-Dependent Cerebral Amyloid Angiopathy and Spontaneous Hemorrhage in Amyloid Precursor Protein Transgenic Mice
John D. Fryer, Jennie W. Taylor, Ronald B. DeMattos, Kelly R. Bales, Steven M. Paul, Maia Parsadanian, David M. Holtzman
Journal of Neuroscience 27 August 2003, 23 (21) 7889-7896; DOI: 10.1523/JNEUROSCI.23-21-07889.2003
Jump to section
Keywords
Responses to this article
Jump to comment:
No eLetters have been published for this article.