Table 3.

The main results were replicated using four different sets of models that included different sets of predictors

ModelErrorDifferenceBalanced errorBalanced differenceCongruent balanced error and differenceInteraction of error and difficultySlope (βed)
0–300 ms
    Full2834222210161.43
24%30%19%19%9%14%p < 10−5
    Drop3238273113181.44
28%33%23%27%11%16%p < 10−5
    Base3337212810181.49
29%32%18%24%9%15%p < 10−6
    Reduced3537193318201.53
30%32%17%29%16%17%p < 10−5
0–200 ms
    Full273618247131.63
23%31%16%21%6%11%p < 10−4
    Drop283819288161.62
24%33%17%24%7%14%p < 10−5
    Base293820288211.61
25%33%17%24%7%18%p < 10−5
    Reduced293618247161.74
25%31%16%21%6%14%p < 10−5
−400 to −200 ms
    Full673808−1.73
5%6%3%7%0%7%Ns
    Drop744809−1.68
6%3%3%7%0%8%Ns
    Base654708−0.90
5%4%3%6%0%7%p < 0.05
    Reduced475607−1.05
3%6%4%5%0%6%p < 0.05
  • All numbers indicate how many cells passed a corresponding statistical test at α = 0.05. Bold indicate that this value is significantly higher than expected by chance (binomial test, p < 0.01, p0 = α; df = 114; p0 = α2/2 for the conjunction analysis of error and difficulty). The same analyses were conducted using two additional time epochs from 0 to 200 ms and from −400 to −200 ms. The 0–200 ms epoch replicates the main findings and shows that similar signals can also be found closer to saccade onset. The −400 to −200 ms epoch provides a negative control and shows that, as expected, none of the findings can be explained by elevated false-positive rate of any of the tests. Ns, Not significant.