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Non-prion-type transmission in A53T α-synuclein transgenic mice: a normal component of spinal homogenates from naïve non-transgenic mice induces robust α-synuclein pathology

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Acknowledgments

This study was supported by NINDS R01-NS089622 (BG) and R01-NS092788 (DB).

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Authors and Affiliations

  1. Department of Neuroscience, College of Medicine University of Florida, Gainesville, FL, 32610, USA

    Amanda N. Sacino, Jacob I. Ayers, Mieu M. T. Brooks, Paramita Chakrabarty, Vincent J. Hudson III, Jasie K. Howard, Todd E. Golde, Benoit I. Giasson & David R. Borchelt

  2. Center for Translational Research in Neurodegenerative Disease, College of Medicine University of Florida, Gainesville, FL, 32610, USA

    Amanda N. Sacino, Jacob I. Ayers, Mieu M. T. Brooks, Paramita Chakrabarty, Vincent J. Hudson III, Jasie K. Howard, Todd E. Golde, Benoit I. Giasson & David R. Borchelt

  3. McKnight Brain Institute, College of Medicine University of Florida, Gainesville, FL, 32610, USA

    Jacob I. Ayers, Paramita Chakrabarty, Todd E. Golde, Benoit I. Giasson & David R. Borchelt

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  1. Amanda N. Sacino
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  2. Jacob I. Ayers
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  3. Mieu M. T. Brooks
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  5. Vincent J. Hudson III
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  6. Jasie K. Howard
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  7. Todd E. Golde
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  8. Benoit I. Giasson
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  9. David R. Borchelt
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Corresponding authors

Correspondence to Benoit I. Giasson or David R. Borchelt.

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Sacino, A.N., Ayers, J.I., Brooks, M.M.T. et al. Non-prion-type transmission in A53T α-synuclein transgenic mice: a normal component of spinal homogenates from naïve non-transgenic mice induces robust α-synuclein pathology. Acta Neuropathol 131, 151–154 (2016). https://doi.org/10.1007/s00401-015-1505-1

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  • DOI: https://doi.org/10.1007/s00401-015-1505-1

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