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Fluid intake, what's dopamine got to do with it?
2021, Physiology and BehaviorCatecholaminergic axons in the neocortex of adult mice regrow following brain injury
2020, Experimental NeurologyDopaminergic modulation of pain signals in the medial prefrontal cortex: Challenges and perspectives
2019, Neuroscience LettersCitation Excerpt :However, many studies have implicated DA in aversion and pain [18,20,60]. Dopaminergic neurons in the VTA make reciprocal projections with the mPFC [12,23,24] and play important modulating roles in fine tuning the neuronal activities and inducing plasticity [33,84]. Behavioral studies have documented effects of dopaminergic modulation in the mPFC in working memory and attention [38,45].
Destruction of noradrenergic terminals increases dopamine concentration and reduces dopamine metabolism in the medial prefrontal cortex
2018, Behavioural Brain ResearchCitation Excerpt :Our earlier data showed that DA levels were significantly reduced in the brain tissue after the 6-OHDA lesion with the protection of NE fibers, while 6-OHDA lesion alone decreased the NE concentration, but did not cause decrease in the DA level [25]. Based on these [25] and other findings in the literature [41–45], two types of bilateral neurochemical lesion were applied to destroy NE terminals in the medial PFC. In one group of animals, 6-hydroxy-DOPA (6-OHDOPA), the ortho-hydroxylated derivate of L-DOPA was injected into the medial PFC. The 6-OHDOPA is a cytotoxic amino acid, which is taken up by NE terminals [47–51]. In another group of animals, the potent DA reuptake blocker GBR 12935 was injected before the administration of neurotoxin 6-OHDA to enhance the selectivity of the lesion for NE terminals.
Cortical determinants of goal-directed behavior
2018, Goal-Directed Decision Making: Computations and Neural Circuits