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2021, Biochemical and Biophysical Research CommunicationsCitation Excerpt :The NET regulates noradrenergic signaling in various physiological processes, including behavior, mood, cognition, and regulating blood pressure and heart rate [8,9]. Although NET is present on cells synthesizing NE such as sympathetic nerve cells and adrenal chromaffin cells [4,5,7], whether mammary epithelial cells express NET is unknown. In this study, we evaluated the expression and localization of the NET in human mammary epithelial cells and mouse mammary tissue.
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2018, Comprehensive Toxicology: Third EditionEffects of exposure to amitraz on noradrenaline, serotonin and dopamine levels in brain regions of 30 and 60 days old male rats
2013, ToxicologyCitation Excerpt :The present study evaluated in rats the effects of amitraz on norepinephrine, serotonin and dopamine and metabolites levels, as well as the neurotransmitter rate (turnover), a measure of presynaptic neuronal activity (Dam et al., 1999; Seider and Slotkin, 1990) in five brain regions (hypothalamus, midbrain, hippocampus, striatum and prefrontal cortex), major areas of monoaminergic systems involved in cognitive performance, learning and memory, and motor activity, which could be targets for amitraz. On the basis of current reports suggesting that innervation of monoaminergic neurons in the brain progresses with increasing age as well as developmental changes in catecholamine biosynthetic enzymes (Nomura et al., 1976; Rinaman, 2001), the present study was carried out using male rats at 30 days of age (immature or pubertal rats) and at 60 days of age (adult rats). The aim of our work is to determine amitraz effects on CNS monoamine levels in a brain regional-, dose- and age-related manner.
The Developing Nervous System
2010, Comprehensive Toxicology, Second EditionBrain noradrenaline changes in rats prenatally exposed to ozone
2010, Environmental Toxicology and PharmacologyCitation Excerpt :However, the body weight of pups at birth was significantly lower (t = 5.01, df = 46, p < 0.0001) from O3-exposed mothers (4.7 ± 0.15 g) as compared to those obtained from non-O3-exposed mothers (6.8 ± 0.26 g), but the weight of the whole brain, cerebellum, cerebral cortex, and pons at 10, 20 and 30 days old did not show significant differences between groups. Our findings that cerebellar NA levels were elevated compared with those of the cerebral cortex in the control groups are in agreement with previous findings by Nomura et al. (1976) and Mason and Fibiger (1979). However, the NA concentration in the cerebellum was significantly depleted in pups with prenatal exposition to O3 at 10, 20, and 30 days old as compared to control group.