The effects of phencyclidine and N-allylnormetazocine on midbrain dopamine neuronal activity
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Potential mechanisms for phencyclidine/ketamine-induced brain structural alterations and behavioral consequences
2020, NeuroToxicologyCitation Excerpt :Because dopamine is implicated in the etiology of schizophrenia and in drug abuse, the interaction of PCP or ketamine with dopaminergic and other neurotransmitter systems has been broadly investigated (Lodge and Mercier, 2015; Vickroy and Johnson, 1983). PCP inhibits dopamine uptake and increases dopamine release (Garey and Heath, 1976; Vickroy and Johnson, 1980, 1982), and the behavioral actions of PCP could be reversed by functional antagonists of dopaminergic transmission (Freeman and Bunney, 1984). Developing neurons have the highest expression of PSA, which is thought to promote neural plasticity, and thereby, possibly facilitate the migration of neural precursors and assist in axonal pathfinding and synaptogenesis.
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2016, Neuropathology of Drug Addictions and Substance Misuse Volume 2: Stimulants, Club and Dissociative Drugs, Hallucinogens, Steroids, Inhalants and International AspectsDehydroepiandrosterone alleviates copulatory disorder induced by social stress in male rats
2006, Journal of Sexual MedicineCitation Excerpt :Sigma 1 receptors are widely distributed in the central nervous system of rats, including regions responsible for copulatory behavior [31]. Autoradiographic binding was reduced by systemic injection of 6‐hydroxydopamine [31], while neuronal activity of midbrain dopaminergic neurons (A9 and A10) [32,33] and extracellular dopamine in the striatum [34] were affected by administration of sigma ligands. Patrick et al. reported that two sigma ligands, 1,3‐di‐o‐tolylguanidine and pentazocine, produced a significant (30–50%) increase in extracellular domapine [34].