Dopamine uptake during fast-phase endogenous dopamine release from mouse striatal synaptosomes
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The evolution of dystonia-like movements in TOR1A rats after transient nerve injury is accompanied by dopaminergic dysregulation and abnormal oscillatory activity of a central motor network
2021, Neurobiology of DiseaseCitation Excerpt :In our previously analyzed Tor1a+/− mouse model, administration of levodopa worsened DLM while treatment with a DA synthesis blocker reduced DLM, suggesting that a striatal overavailability of DA could trigger abnormal downstream signaling (Ip et al., 2016), similar to the dopaminergic mechanisms causing levodopa-induced dyskinesia in Parkinson's disease (Cenci and Crossman, 2018). Membrane depolarization induced by local potassium infusion is known to increase DA efflux; previous studies have revealed a reduction in striatal DA uptake in depolarized conditions due to internalization of DAT as a possible mechanism (Richardson et al., 2016; Woodward et al., 1986). It's known that torsinA influences DAT expression on the cell surface, which would in turn affect DA uptake and extracellular DA homeostasis (Torres et al., 2004).
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1993, Neurochemistry InternationalThe putative polyamine antagonists ifenprodil and SL 82.0715 enhance dopamine efflux from rat striatal slices independent of NMDA receptor activation
1992, European Journal of PharmacologyActivity-dependent recruitment of endogenous glutamate for exocytosis
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