Demonstration of neurofibrillary tangles in parvalbumin-immunoreactive interneurones in the cerebral cortex of Alzheimer-type dementia brain
References (17)
- et al.
Loss of parvalbumin-immunoreactive neurones from cortex in Alzheimer-type dementia
Brain Res.
(1987) Measurement of facilitated calcium diffusion by a soluble calcium-binding protein
Biochem. Biophys. Acta
(1984)- et al.
Glutamate dysfunction in Alzheimer's disease: an hypothesis
Trends Neurosci.
(1987) - et al.
Cortical neuronal counts in normal elderly controls and demented patients
Neurobiol. Aging
(1983) - et al.
Plaques, tangles and dementia: a study
J. Neurol. Sci.
(1982) - et al.
Calcium-binding protein parvalbumin as a neuronal marker
Nature
(1981) Parvalbumin, an intracellular calcium-binding protein; distribution, properties and possible roles in mammalian cells
Experientia
(1984)- et al.
Alzheimer's neurofibrillary changes
Arch. Neurol.
(1961)
There are more references available in the full text version of this article.
Cited by (23)
The dentate gyrus in Alzheimer's disease
2007, Progress in Brain ResearchCitation Excerpt :Interneurons seem to be very resistant, perhaps because of their high content of calcium-binding proteins such as parvalbumin or calretinin (Braak et al., 1991; Nitsch and Ohm, 1995; Freund and Buzsaki, 1996). However, an obligatory absence cannot be presumed because in frontal cortex 4–6% parvalbumin-containing neurons also reacted with an antibody raised against tau from tangles (Iwamoto and Emson, 1991). Others have reported only a 0–0.7% subpopulation of parvalbumin-containing neurons of the superior frontal gyrus which may have tangles (Sampson et al., 1997).
m-calpain (calcium-activated neutral proteinase) in alzheimer's disease brains
1998, Neuroscience LettersAge-related changes in parvalbumin- and GABA-immunoreactive cells in the rat septum
1995, Neurobiology of Aging
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