Reversible and irreversible neuronal damage caused by excitatory amino acid analogues in rat cerebellar slices
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Analyses of smooth endoplasmic reticulum of cerebellar parallel fibers in aging, ethanol-fed rats
2005, AlcoholCitation Excerpt :Ethanol-related dilation of SER in parallel fibers has not been assessed. The plausibility of dilation of parallel fiber SER after chronic ethanol consumption, however, is supported by the fact that chronic (48 h) ethanol treatment up-regulates N-methyl-d-aspartate receptors (Chen et al., 1999) and dilation of granule neuron endoplasmic reticulum occurs after prolonged exposure of cerebellar slices to N-methyl-d-aspartate (Hajós et al., 1986). The finding of SER dilation in parallel fibers, taken with the previous report of SER dilation in Purkinje neuron dendrites (Dlugos & Pentney, 2000), would seem to indicate that SER dilation is an overall effect of ethanol on neuronal processes in the aging cerebellum.
Prepulse inhibition in rats with temporary inhibition/inactivation of ventral or dorsal hippocampus
2002, Pharmacology Biochemistry and BehaviorCalpain activation and not oxidative damage mediates L-2- chloropropionic acid-induced cerebellar granule cell necrosis
1997, Toxicology and Applied Pharmacology
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Permanent address: 1st Department of Anatomy, Semmelweis University Medical School, 1450 Tüzoltóutca 58, Budapest IX, Hungary.