A syntaxin-related protein controls acetylcholine release by different mechanisms inAplysia
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2005, Microvascular ResearchRole of syntaxin 1A on serotonin transporter expression in developing thalamocortical neurons
2002, International Journal of Developmental NeuroscienceCitation Excerpt :Syntaxin 1A was originally characterized as a component of the machinery involved in transmitter release (Bennett et al., 1993; Söllner et al., 1993), and is a key component in the SNARE hypothesis of vesicle trafficking and fusion (Südhof, 1995; Rothman, 1996). More recently, syntaxin 1A has been shown to directly regulate Ca2+ channels (Sheng et al., 1994; Bezprozvanny et al., 1995; Smirnova et al., 1995; Wiser et al., 1996; Sutton et al., 1999; Jarvis et al., 2000), cystic fibrosis Cl− channels (Naren et al., 1997, 1998), K+ channels (Fili et al., 2001) and epithelial Na+ channels (Qi et al., 1999; Saxena et al., 1999). Syntaxin 1A also interacts with members of the family of Na+ and Cl− dependent neurotransmitter transporters (Quick et al., 1997; Beckman et al., 1998; Geerlings et al., 2000).
Bidirectional modulation of transmitter release by calcium channel/syntaxin interactions in vivo
2007, Journal of NeuroscienceRegulation of syntaxin 1A-munc18 complex for SNARE pairing in HEK293 cells
2004, Journal of Physiology