Short communicationReperfusion following focal cerebral ischemia alters distribution of neuronal cells with DNA fragmentation in mice
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Acknowledgements
The present study was supported by National Institutes of Health Grants NS 14543, NS 25372 and NOI-NS-52334. The authors thank S. Chen, L. Reola and B. Calagui for their technical assistance and C. Christensen for her editorial assistance.
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Non-NMDA mechanism in the inhibition of cellular apoptosis and memory impairment induced by repeated ischemia in rats
2004, Brain ResearchCitation Excerpt :One of the important properties which the experimental models of cerebral ischemia should be characterized by is the resemblance of their underlying pathological changes to those taking place in the cerebrovascular diseases in which delayed neuronal cell death, possibly apoptosis associated with DNA fragmentation [9,22,24], is the hall mark change taking place.
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