Mitochondrial function and NMDA receptor activation: mechanisms of secondary excitotoxicity

Funct Neurol. 1999 Jul-Sep;14(3):171-84.
No abstract available

Publication types

  • Review

MeSH terms

  • Animals
  • Calcium / physiology
  • Cell Death / physiology*
  • Energy Metabolism / physiology
  • Glutamic Acid / physiology
  • Humans
  • Mitochondria / physiology*
  • Neurodegenerative Diseases / physiopathology*
  • Neurons / physiology
  • Neurotoxins / metabolism*
  • Receptors, N-Methyl-D-Aspartate / physiology*

Substances

  • Neurotoxins
  • Receptors, N-Methyl-D-Aspartate
  • Glutamic Acid
  • Calcium