Limiting stroke-induced damage by targeting an acid channel

N Engl J Med. 2005 Jan 6;352(1):85-6. doi: 10.1056/NEJMcibr045010.
No abstract available

MeSH terms

  • Acid Sensing Ion Channels
  • Acidosis / drug therapy
  • Acidosis / metabolism*
  • Animals
  • Brain Ischemia / drug therapy
  • Brain Ischemia / metabolism*
  • Calcium / metabolism
  • Calcium Signaling / drug effects
  • Disease Models, Animal
  • Membrane Proteins / antagonists & inhibitors
  • Membrane Proteins / metabolism*
  • Mice
  • Nerve Degeneration / drug therapy
  • Nerve Degeneration / metabolism
  • Nerve Tissue Proteins / antagonists & inhibitors
  • Nerve Tissue Proteins / metabolism*
  • Neuroprotective Agents / pharmacology
  • Neuroprotective Agents / therapeutic use
  • Receptors, N-Methyl-D-Aspartate / metabolism
  • Sodium Channels / metabolism*
  • Stroke / drug therapy
  • Stroke / metabolism

Substances

  • Acid Sensing Ion Channels
  • Membrane Proteins
  • Nerve Tissue Proteins
  • Neuroprotective Agents
  • Receptors, N-Methyl-D-Aspartate
  • Sodium Channels
  • Calcium